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Diabetes Care 29:468 2006
DOI: 10.2337/diacare.29.02.06.dc05-1484
© 2006 by the American Diabetes Association
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Letters: Observations

Association of Serum Fetuin-A With Insulin Resistance in Type 2 Diabetic and Nondiabetic Subjects

Katsuhito Mori, MD, Masanori Emoto, MD, Hisayo Yokoyama, MD, Takahiro Araki, MD, Megumi Teramura, MD, Hidenori Koyama, MD, Tetsuo Shoji, MD, Masaaki Inaba, MD and Yoshiki Nishizawa, MD

Department of Metabolism, Endocrinology and Molecular Medicine, Osaka City University Graduate School of Medicine, Osaka, Japan

Address correspondence to Katsuhito Mori, MD, Department of Metabolism, Endocrinology and Molecular Medicine, Osaka City University Graduate School of Medicine, 1-4-3, Asahi-machi, Abeno-ku, Osaka 545-8585, Japan. E-mail: ktmori@med.osaka-cu.ac.jp

The first 20% of the full text of this article appears below.

Fetuin-A ({alpha}2-Heremans Schmid glycoprotein) is a circulating glycoprotein that can inhibit insulin receptor autophosphorylation and subsequent downstream signaling in vitro (1, 2). Recently, it has been reported (3) that fetuin-A–deficient mice demonstrate enhanced insulin sensitivity. These data indicate that fetuin-A might be a negative regulator of insulin signaling. However, the physiological significance of fetuin-A in insulin resistance in humans remains unclear.

To address this, we investigated the relationship of serum fetuin-A levels and insulin resistance in nondiabetic (n = 160) and type 2 diabetic (n = 161) subjects. Serum fetuin-A was measured by an enzyme-linked . . . [Full Text of this Article]


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