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Glucose, Lipid, and Blood Pressure Control in Australian Adults With Type 2 Diabetes

The 1999–2000 AusDiab

  1. Thomas M. Kemp, FRCP1,
  2. Elizabeth L.M. Barr, MPH2,
  3. Paul Z. Zimmet, AO, MD, PHD2,
  4. Adrian J. Cameron, MPH2,
  5. Timothy A. Welborn, AO, PHD3,
  6. Stephen Colagiuri, MD4,
  7. Patrick Phillips, MD5,
  8. Jonathan E. Shaw, MD2 and
  9. on behalf of the AusDiab Steering Committee
  1. 1Department of Medicine, Dewsbury & District Hospital, Dewsbury, West Yorkshire, U.K.
  2. 2International Diabetes Institute, Caulfield, Victoria, Australia
  3. 3Department of Medicine, University of Western Australia, Nedlands, Western Australia, Australia
  4. 4Department of Endocrinology, Diabetes and Metabolism, The Prince of Wales Hospital, Randwick, New South Wales, Australia
  5. 5Endocrinology Unit, North Western Adelaide Health Service, Queen Elizabeth Hospital, Woodville, South Australia, Australia
  1. Address correspondence and reprint requests to Elizabeth L.M. Barr, International Diabetes Institute, 250 Kooyong Rd., Caulfield, Victoria 3162, Australia. E-mail: lbarr{at}idi.org.au

The risk of diabetes complications can be reduced by tight control of blood glucose (1), serum lipids (2), and blood pressure (3,4). However, evidence from a limited number of studies (5–9) indicates that many people with type 2 diabetes do not achieve recommended targets for these factors. We examined levels of glucose, lipid, and blood pressure control in participants with type 2 diabetes taking part in the national population-based Australian Diabetes, Obesity, and Lifestyle Study (AusDiab) (10) conducted during 1999–2000.

RESEARCH DESIGN AND METHODS

AusDiab was a national population-based survey of the general population and has been described in detail earlier (10). Diagnosis of diabetes was based on self-reported physician diagnosis of diabetes confirmed either by self-reported use of hypoglycemic drugs or results from a 75-g oral glucose tolerance test (11). Participants who started insulin treatment within 2 years of diagnosis were classified as having type 1 diabetes (if diabetes onset was at age ≥40 years; BMI also had to be <27 kg/m2). All other cases were classified as type 2 diabetes.

Fasting (≥9 h) serum total cholesterol, LDL and HDL cholesterol, and triglycerides were measured (Olympus AU600 analyzer; Olympus Optical, Tokyo, Japan). Total glycated hemoglobin analysis used high-performance liquid chromatography (Bio-Rad Variant Hemoglobin Testing System; Bio-Rad, Hercules, CA) with standardized conversion to HbA1c values …

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