Table 1—

Multivariable-adjusted risk estimates for the association between baseline dietary glycemic index and glycemic load and the risk of developing type 2 diabetes, stratified by abdominal obesity at baseline and change in waist during follow-up, IRAS I and II, n = 892

Casesβ (P value)OR (95% CI)
P value
1st tertile2nd tertile3rd tertile
Glycemic index
    Abdominal obesity*
Yes74−0.0035 (0.90)1.000.82 (0.39–1.72)0.84 (0.40–1.79)0.65
No720.0517 (0.06)1.001.42 (0.68–2.97)1.90 (0.89–4.00)0.10
    Change in waist§
Decrease230.0404 (0.40)1.00
Stable36−0.0678 (0.14)1.000.85 (0.30–2.42)0.49 (0.14–1.66)0.25
Increase870.0571 (0.04)1.001.32 (0.65–2.65)1.70 (0.84–3.47)0.14
Glycemic load
    Abdominal obesity*
Yes74−0.0017 (0.74)1.001.20 (0.58–2.48)0.82 (0.39–1.75)0.89
No72−0.0019 (0.70)1.001.31 (0.67–2.57)1.14 (0.57–2.26)0.66
    Change in waist§
Decrease23−0.0007 (0.94)1.00
Stable36−0.0131 (0.10)1.00
        Increase87−0.0006 (0.90)1.000.81 (0.43–1.53)0.76 (0.39–1.49)0.42
  • *

    * Waist circumference >102 cm for men and >88 cm for women.

  • Adjusted for age, ethnicity/clinic, baseline BMI, family history of diabetes, smoking status, glucose tolerance status, education, and energy intake (by residual method).

  • Decrease: change in waist ≤−2 cm; stable: change in waist ±2 cm; increase: change in waist ≥2 cm.

  • §

    § Adjusted for age, ethnicity/clinic, baseline BMI, baseline waist circumference, family history of diabetes, smoking status, glucose tolerance status, education, and energy intake (by residual method).

  • Tertile-specific ORs were not estimated if the reference category comprised <10 subjects.